[61]
44 TABLE 21. ENDOMETRIAL (UTERINE CORPUS) CANCER INCIDENCE BY ...
[13,7 KB]
From [www.state.nj.us] Last viewed: 07.09.2006
44
TABLE 21.
ENDOMETRIAL ( UTERINE CORPUS) CANCER INCIDENCE BY COUNTY, WHITE FEMALES,
NEW JERSEY - 1986-1996
1
COUNTY
1986
1987
1988
1989
1990
1991
1992
1993
1994
1995
1996
(Prelim.)
No. Rate No. Rate No. Rate No. Rate No. Rate No. Rate No. Rate No. Rate No. Rate No. Rate No. Rate
Atlantic
24 19.9
26 22.6
23 17.4
15 14.2
27 22.9
33 24.5
30 24.2
37 31.2
35 27.9
26 21.4
22 18.0
Bergen
123 20.6 136 24.1 121 20.6 115 20.2
98 16.7 161 29.2 135 23.9 125 22.3 139 24.6 126 22.6 138 23.9
Burlington
26 13.7
41 22.1
31 15.9
40 19.2
42 21.3
38 19.3
35 18.0
59 28.9
57 26.8
57 28.2
47 22.3
Camden
50 18.8
43 15.1
50 19.3
61 22.2
61 23.0
61 23.8
55 21.9
45 18.3
55 22.0
...
[62]
UTERINE CANCER
[57,9 KB]
From [www.vandenberg.af.mil] Last viewed: 07.09.2006
Copyright © 1998 by W.B. Saunders Company.
All rights reserved.
510
A
BASIC INFORMATION
DESCRIPTION
Cancer of the endometrium (lining of the uterus). It
usually affects postmenopausal women ages 50 to 60.
FREQUENT SIGNS AND SYMPTOMS
Early stages:
• Bleeding or spotting, especially after sexual inter-
course. This often occurs after menstrual activity has
ceased for 12 months or more. A watery or blood-
streaked vaginal discharge may precede bleeding or
spotting.
• Enlarged uterus. It is sometimes a large enough mass
to be felt externally.
Later stages:
• Spread to other organs, causing abdominal pain, chest
pain and weight loss.
CAUSES
Unknown. Appears to be linked to several predisposing
factors listed in Risk.
RISK INCREASES WITH
• Diabetes mellitus.
• Obesity.
• High blood pressure.
• Use of estrogen without also ...
[63]
Monitoring of Rheologic Variables During Postoperative High-Dose ...
From [cat.sagepub.com] Last viewed: 07.09.2006
Page 2
Cancer of the uterus in England 1996 to 2004.
A geographical profile of mortality.
Authors: Michael Goldacre, David Yeates, Leicester Gill,
Myfanwy Griffith, Henry McGuinness, Daniel Meddings
Published by: Unit of Health-Care Epidemiology, Oxford University,
and South East England Public Health Observatory, 2005
This document provides a geographical profile of mortality from cancer of
the uterus in England. The geographical areas covered are the standard
local authority areas of England. The period covered is January 1 1996 to
December 31 2004. The data are analysed from mortality files supplied to
the Public Health Observatories by the Office for National Statistics. This
analysis includes mortality data about females of all ages.
The map, chart and table show standardised mortality ratios (SMRs). The
data in the map are shown as quintiles of local authorities, ...
[64]
Association of Family History of Cervical, Ovarian, and Uterine ...
[896,9 KB]
From [cebp.aacrjournals.org] Last viewed: 07.09.2006
Copyright © 1998 by W.B. Saunders Company.
All rights reserved.
510
A
BASIC INFORMATION
DESCRIPTION
Cancer of the endometrium (lining of the uterus). It
usually affects postmenopausal women ages 50 to 60.
FREQUENT SIGNS AND SYMPTOMS
Early stages:
• Bleeding or spotting, especially after sexual inter-
course. This often occurs after menstrual activity has
ceased for 12 months or more. A watery or blood-
streaked vaginal discharge may precede bleeding or
spotting.
• Enlarged uterus. It is sometimes a large enough mass
to be felt externally.
Later stages:
• Spread to other organs, causing abdominal pain, chest
pain and weight loss.
CAUSES
Unknown. Appears to be linked to several predisposing
factors listed in Risk.
RISK INCREASES WITH
• Diabetes mellitus.
• Obesity.
• High blood pressure.
• Use of estrogen without also ...
[65]
Relationship between the cervical uterine cancer evolution and ...
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[66]
Age-related characteristics of uterine cancer mortality in Japan
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[67]
Chemoradiotherapy for uterine cancer: current status and perspectives
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[68]
Heat-stable alkaline phosphatase in uterine cancer, with special ...
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[69]
Intravaginal bacterial flora in patients with uterine cervical ...
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[70]
Survival prediction using artificial neural networks in patients ...
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[71]
Differences between Black and White patients with cancer of the ...
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[72]
An unusual case of uterine tube cancer (transitional cell ...
[19,7 KB]
From [www.springerlink.com] Last viewed: 07.09.2006
UTERINE SARCOMA - CANCER SITES
UTERINE SARCOMA
What Is Cancer ?
Cancer develops when cells in a part of the body begin to grow out of control. Although there are many kinds of
cancer , they all start because of out-of-control growth of abnormal cells.
Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person's life, normal
cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide
only to replace worn-out or dying cells and to repair injuries.
Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they
outlive normal cells and continue to form new abnormal cells.
Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities.
Most of the time when DNA becomes damaged the body is able to repair it. ...
[73]
Neoadjuvant Chemotherapy by Transcatheter Arterial Infusion Method ...
[69,5 KB]
From [www.jvir.org] Last viewed: 07.09.2006
© Kamla-Raj 2003
KEYWORDS Cervix smears; micronuclei; exfoliated
urine cells
ABSTRACT Cytogenetic damage was assessed using
the (MN) test in urothelial cells and cervix smears of
local cervix cancer patients since the genetic end-point
screening for micronuclei provides a measure of both,
chromosome breakage and loss. The MN data were
grouped for stage-types of cancer , age-groups, age-at-
marriage, parity levels and socio-economic status. A
comparison of the results obtained from both the tissues
has revealed that the percent frequency of MNd cells
was elevated in urothelial cells except when the variable
for age-groups of the patients was compared. If validated,
the MN assay in urothelial cells may prove useful for
screening programmes for cervix cancer as besides
scoring effectively for cytogenetic damage, it utilizes a
non-invasive process of sample collection.
INTRODUCTION ...
[74]
Enhanced expression of Mcm proteins in cancer cells derived from ...
[512,8 KB]
From [content.febsjournal.org] Last viewed: 07.09.2006
© Kamla-Raj 2003
KEYWORDS Cervix smears; micronuclei; exfoliated
urine cells
ABSTRACT Cytogenetic damage was assessed using
the (MN) test in urothelial cells and cervix smears of
local cervix cancer patients since the genetic end-point
screening for micronuclei provides a measure of both,
chromosome breakage and loss. The MN data were
grouped for stage-types of cancer , age-groups, age-at-
marriage, parity levels and socio-economic status. A
comparison of the results obtained from both the tissues
has revealed that the percent frequency of MNd cells
was elevated in urothelial cells except when the variable
for age-groups of the patients was compared. If validated,
the MN assay in urothelial cells may prove useful for
screening programmes for cervix cancer as besides
scoring effectively for cytogenetic damage, it utilizes a
non-invasive process of sample collection.
INTRODUCTION ...
[75]
Serum Anti-p53 Antibodies in Uterine and Ovarian Cancer ...
[7,9 KB]
From [content.karger.com] Last viewed: 07.09.2006
REVIEW ARTICLE
CURRENT SCIENCE, VOL. 78, NO. 1, 10 JANUARY 2000
52
Cancer of the uterine cervix and human
papillomavirus infection
B. C. Das*, V. Gopalkrishna, Suresh Hedau and Sanjay Katiyar
Division of Molecular Oncology, Institute of Cytology and Preventive Oncology (ICMR), Maulana Azad Medical College Campus,
Bahadur Shah Zafar Marg, New Delhi 110 002, India
Human papillomaviruses (HPVs) have emerged as the
principal sexually transmitted causal agents in the
development of cancer of the uterine cervix in women. They
also cause a variety of benign lesions, warts, intraepithelial
neoplasia and anogenital, oral and pharyngeal papillomas.
Presently, more than 100 HPV genotypes have been
identified in humans, and about one-third of them have been
sequenced. Of these, while HPV types 16 and 18 are
considered to be the high-risk types, HPV 6 and 11 ...
[76]
Spontaneous Remission of Metastases of Cancer of the Uterine Cervix
[7,9 KB]
From [content.karger.com] Last viewed: 07.09.2006
REVIEW ARTICLE
CURRENT SCIENCE, VOL. 78, NO. 1, 10 JANUARY 2000
52
Cancer of the uterine cervix and human
papillomavirus infection
B. C. Das*, V. Gopalkrishna, Suresh Hedau and Sanjay Katiyar
Division of Molecular Oncology, Institute of Cytology and Preventive Oncology (ICMR), Maulana Azad Medical College Campus,
Bahadur Shah Zafar Marg, New Delhi 110 002, India
Human papillomaviruses (HPVs) have emerged as the
principal sexually transmitted causal agents in the
development of cancer of the uterine cervix in women. They
also cause a variety of benign lesions, warts, intraepithelial
neoplasia and anogenital, oral and pharyngeal papillomas.
Presently, more than 100 HPV genotypes have been
identified in humans, and about one-third of them have been
sequenced. Of these, while HPV types 16 and 18 are
considered to be the high-risk types, HPV 6 and 11 ...
[77]
Uterine Cervical Metastasis of Breast Cancer: A Rare Complication ...
[7,9 KB]
From [content.karger.com] Last viewed: 07.09.2006
REVIEW ARTICLE
CURRENT SCIENCE, VOL. 78, NO. 1, 10 JANUARY 2000
52
Cancer of the uterine cervix and human
papillomavirus infection
B. C. Das*, V. Gopalkrishna, Suresh Hedau and Sanjay Katiyar
Division of Molecular Oncology, Institute of Cytology and Preventive Oncology (ICMR), Maulana Azad Medical College Campus,
Bahadur Shah Zafar Marg, New Delhi 110 002, India
Human papillomaviruses (HPVs) have emerged as the
principal sexually transmitted causal agents in the
development of cancer of the uterine cervix in women. They
also cause a variety of benign lesions, warts, intraepithelial
neoplasia and anogenital, oral and pharyngeal papillomas.
Presently, more than 100 HPV genotypes have been
identified in humans, and about one-third of them have been
sequenced. Of these, while HPV types 16 and 18 are
considered to be the high-risk types, HPV 6 and 11 ...
[78]
Enhanced Expression of Thymidylate Synthase Mediates Resistance of ...
[7,9 KB]
From [content.karger.com] Last viewed: 07.09.2006
REVIEW ARTICLE
CURRENT SCIENCE, VOL. 78, NO. 1, 10 JANUARY 2000
52
Cancer of the uterine cervix and human
papillomavirus infection
B. C. Das*, V. Gopalkrishna, Suresh Hedau and Sanjay Katiyar
Division of Molecular Oncology, Institute of Cytology and Preventive Oncology (ICMR), Maulana Azad Medical College Campus,
Bahadur Shah Zafar Marg, New Delhi 110 002, India
Human papillomaviruses (HPVs) have emerged as the
principal sexually transmitted causal agents in the
development of cancer of the uterine cervix in women. They
also cause a variety of benign lesions, warts, intraepithelial
neoplasia and anogenital, oral and pharyngeal papillomas.
Presently, more than 100 HPV genotypes have been
identified in humans, and about one-third of them have been
sequenced. Of these, while HPV types 16 and 18 are
considered to be the high-risk types, HPV 6 and 11 ...
[79]
Frequent Alterations of the ß-Catenin Protein in Cancer of the ...
[7,9 KB]
From [content.karger.com] Last viewed: 07.09.2006
REVIEW ARTICLE
CURRENT SCIENCE, VOL. 78, NO. 1, 10 JANUARY 2000
52
Cancer of the uterine cervix and human
papillomavirus infection
B. C. Das*, V. Gopalkrishna, Suresh Hedau and Sanjay Katiyar
Division of Molecular Oncology, Institute of Cytology and Preventive Oncology (ICMR), Maulana Azad Medical College Campus,
Bahadur Shah Zafar Marg, New Delhi 110 002, India
Human papillomaviruses (HPVs) have emerged as the
principal sexually transmitted causal agents in the
development of cancer of the uterine cervix in women. They
also cause a variety of benign lesions, warts, intraepithelial
neoplasia and anogenital, oral and pharyngeal papillomas.
Presently, more than 100 HPV genotypes have been
identified in humans, and about one-third of them have been
sequenced. Of these, while HPV types 16 and 18 are
considered to be the high-risk types, HPV 6 and 11 ...
[80]
Inherited susceptibility to uterine leiomyomas and renal cell cancer
[854,9 KB]
From [www.pnas.org] Last viewed: 07.09.2006
Survival prediction using artificial neural networks in patients with uterine cervical
cancer
Masahiro Iinuma, Kenya Murase
Department of Medical Physics and Engineering, Graduate School of Medicine, Osaka University, Osaka, Japan
Abstract
Our purpose of this study was to construct the predict model
using artificial neural networks (ANNs) from radiation
oncology database. Although there is no guarantee that
trained ANNs models reached an error minimum point, we
made 40 models by each combination of hidden nodes (30, 50,
100, and 150). Using the trained ANNs, we predicted the
survival in patients with uterine cervical cancer treated by
radiation therapy. The performance of ANNs was evaluated
using receiver operating characteristic (ROC) analysis by
those maximums and the mean. Az value was the highest at the
number of neuron 150, and the value was maximum ...
[81]
Pulmonary Metastasectomy for Uterine Cervical Cancer: A ...
From [ats.ctsnetjournals.org] Last viewed: 07.09.2006
Age
Sex
Site
Diagnosis
pTNM
Stage
1
70
F
cervix
squamous cell carcinoma
T1aN0M0
I
2
46
F
cervix
squamous cell carcinoma
T1a1N0M0
IA1
3
42
F
cervix
microinvasive squamous cell carcinoma
T1a1N0M0
IA1
4
57
F
cervix
squamous cell carcinoma in situ
TisN0M0
0
5
33
F
cervix
microinvasive squamous cell carcinoma
T1a1N0M0
IA1
6
35
F
cervix
squamous cell carcinoma in situ
TisN0M0
0
7
48
F
cervix
microinvasive squamous cell carcinoma
T1a1N0M0
IA1
8
39
F
cervix
microinvasive squamous cell carcinoma
T1N0M0
I
9
58
F
cervix
microinvasive squamous cell carcinoma
T1N0M0
I
10
53
F
cervix
squamous cell carcinoma in situ
TisN0M0 ...
[82]
Overexpression of HER-2/Neu in Uterine Serous Papillary Cancer
[324,1 KB]
From [clincancerres.aacrjournals.org] Last viewed: 07.09.2006
Age
Sex
Site
Diagnosis
pTNM
Stage
1
70
F
cervix
squamous cell carcinoma
T1aN0M0
I
2
46
F
cervix
squamous cell carcinoma
T1a1N0M0
IA1
3
42
F
cervix
microinvasive squamous cell carcinoma
T1a1N0M0
IA1
4
57
F
cervix
squamous cell carcinoma in situ
TisN0M0
0
5
33
F
cervix
microinvasive squamous cell carcinoma
T1a1N0M0
IA1
6
35
F
cervix
squamous cell carcinoma in situ
TisN0M0
0
7
48
F
cervix
microinvasive squamous cell carcinoma
T1a1N0M0
IA1
8
39
F
cervix
microinvasive squamous cell carcinoma
T1N0M0
I
9
58
F
cervix
microinvasive squamous cell carcinoma
T1N0M0
I
10
53
F
cervix
squamous cell carcinoma in situ
TisN0M0 ...
[83]
CEREBRAL META S TASIS OF CERVICAL UTERINE CANCER Report of three cases
[1078,2 KB]
From [www.scielo.br] Last viewed: 06.07.2006
Arq Neuropsiquiatr 2006;64(2-A):300-302
D e p a rtment of Neuro s u rg e ry, Hospital Nossa Senhora das Graças, Curitiba PR, Brazil:
1
Resident in Neuro s u rg e ry;
2
N e u ro s u rg e o n ;
3
Neurosurgeon, Federal University of Sergipe (Aracaju SE, Brasil);
4
Neurosurgeon Director of the Residency Program.
Received 7 June 2005, received in final form 16 January 2006. Accepted 23 January 2006.
D r. Joacir Graciolli Cord e i ro - Rua Alcides Munhoz 433 - Neuro c i ru rgia - 80810-040 Curitiba PR - Brasil. E-mail: joacirg c @ h o t m a i l . c o m
CEREBRAL META S TASIS OF CERVICAL UTERINE CANCER
Report of three cases
Joacir Graciolli Cordeiro
1
, Daniel Monte-Serrat Prevedello
2
,
Léo Fernando da Silva Ditzel
2
, Carlos Umberto Pereira
3
, João Cândido Araújo ...
[84]
GEN - UTERINE CANCER
[53,1 KB]
From [www.cedars-sinai.com] Last viewed: 06.07.2006
Approximately 34,000 American women
are diagnosed with uterine cancer each
year. In most instances, the cause is
unknown, but for one in 10 women with
uterine cancer , an inherited predisposition
may be involved. Genetic risk
assessment for uterine cancer should be
considered if a woman:
s
Has been diagnosed with uterine cancer
at an early age (younger than 50)
s
Has been diagnosed with uterine cancer
and another cancer , such as colon,
ovarian, stomach or bile duct
s
Has a history of colon polyps before
age 40
s
Has family members with any of the
above
Of hereditary uterine cancers,
approximately 50 to 70 percent are
associated with the hereditary
nonpolyposis colorectal cancer (HNPCC)
syndrome, also known as Lynch
syndrome. HNPCC is caused by the
inheritance of a predisposing mutation ...
[85]
Trends in Cancer Mortality in Queensland : Uterine cancer
[5,5 KB]
From [foodsafetymatters.gov.au] Last viewed: 06.07.2006
Trends in Cancer Mortality in
Queensland
Uterine cancer
Rate per 100,000 population
Mortality of Uterine cancer ,
among Females, Queensland, 1982 to 2002
Female
0
3
6
9
12
15
Year
1982
1984
1986
1988
1990
1992
1994
1996
1998
2000
2002
Note:
Rates are age standardised to the Australian population as at 30 June 2001. Data are reported by year of
diagnosis. Uterine cancer is defined by the ICD-0-2 codes of C54
Source:
Queensland Cancer Registry, Queensland Health and Queensland Cancer Fund. Generated by Epidemiology
Services Unit, Queensland Health
For more
information:
Health Status Indicators for Queensland
Queensland Health Cancer Publications (http://www.health.qld.gov.au/hic/default.asp)
Queensland Cancer Fund (http://www.qldcancer.com.au/) ...
[86]
Trends in Cancer Mortality in Queensland : Uterine cancer Page 1 ...
[5,5 KB]
From [www.health.qld.gov.au] Last viewed: 06.07.2006
Trends in Cancer Mortality in
Queensland
Uterine cancer
Rate per 100,000 population
Mortality of Uterine cancer,
among Females, Queensland, 1982 to 2002
Female
0
3
6
9
12
15
Year
1982
1984
1986
1988
1990
1992
1994
1996
1998
2000
2002
Note:
Rates are age standardised to the Australian population as at 30 June 2001. Data are reported by year of
diagnosis. Uterine cancer is defined by the ICD-0-2 codes of C54
Source:
Queensland Cancer Registry, Queensland Health and Queensland Cancer Fund. Generated by Epidemiology
Services Unit, Queensland Health
For more
information:
Health Status Indicators for Queensland
Queensland Health Cancer Publications (http://www.health.qld.gov.au/hic/default.asp)
Queensland Cancer Fund (http://www.qldcancer.com.au/)
...
[87]
GEN - UTERINE CANCER
From [csmc.edu] Last viewed: 06.07.2006
Approximately 34,000 American women
are diagnosed with uterine cancer each
year. In most instances, the cause is
unknown, but for one in 10 women with
uterine cancer , an inherited predisposition
may be involved. Genetic risk
assessment for uterine cancer should be
considered if a woman:
s
Has been diagnosed with uterine cancer
at an early age (younger than 50)
s
Has been diagnosed with uterine cancer
and another cancer , such as colon,
ovarian, stomach or bile duct
s
Has a history of colon polyps before
age 40
s
Has family members with any of the
above
Of hereditary uterine cancers,
approximately 50 to 70 percent are
associated with the hereditary
nonpolyposis colorectal cancer (HNPCC)
syndrome, also known as Lynch
syndrome. HNPCC is caused by the
inheritance of a predisposing mutation ...
[88]
Cancer Incidence by Remoteness Category in Queensland : Uterine cancer
[5,4 KB]
From [www.health.qld.gov.au] Last viewed: 25.06.2006
Cancer Incidence by Remoteness
Category in Queensland
Uterine cancer
0
5
10
15
20
25
30
Rate per 100,000 population
Avg No.
per Year
Incidence of Uterine cancer among Females,
by ARIA+ Category, Queensland,
Five Year Average, 1998 to 2002
16.2
151
Major City
ARIAplus Category
16.6
85
Inner Regional
14.5
41
Outer Regional
16.1
3
Remote
13.9
2
Very remote
Note:
Rates are age standardised to the Australian population as at 30 June 2001. Numbers and rates are averaged
over five years, and based on place of usual residence at time of diagnosis. The range shown by the orange line
corresponds to the 95% confidence interval for the estimated rate. Uterine cancer is defined by the ICD-0-2
codes of C54 .
Source:
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